Harris Ripps Memorial At ARVO

Harris Ripps

At this year’s ARVO meeting in Denver, please join us for a session honoring the memory of Dr. Harris Ripps.

Tuesday 1:00 pm – 2:30 pm

Room 708/710/712

Dr. Harris Ripps (1927 – 2014), Proctor Medal winner and past ARVO President, devoted his scientific career to studies on the retina and on causes of visual loss in inherited retinal diseases. He made significant contributions in many areas of vision research, including the kinetics of visual pigment bleaching and regeneration, electrical and chemical communication among retinal neurons and glia, and the cellular mechanisms of retinal degeneration. This memorial session will celebrate Dr. Ripps’ long time vision research career with talks by several his colleagues and students. The audience is welcome to contribute remarks during the open period of the session.

Session chair: John Dowling, Ph.D., Department of Molecular and Cellular Biology, Harvard University


Bradford Ripps, O.D., Total Eyecare, New Jersey
Richard Chappell, Ph.D., Marine Biological Laboratory, Massachusetts
David Pepperberg, Ph.D., Department of Ophthalmology and Visual Sciences, University of Illinois at Chicago
Robert Paul Malchow, Ph.D., Department of Biological Sciences, University of Illinois at Chicago
Haohua Qian, Ph.D., National Eye Institute
Muna Naash, Ph.D., Department of Cell Biology, University of Oklahoma Health Sciences Center
John O’Brien, Ph.D., Department of Ophthalmology and Visual Science, University of Texas
Wen Shen, Ph.D., Department of Biomedical Science, Florida Atlantic University


Seminar: Cellular Response Associated to Retinal Diseases and Therapeutic Approaches

Nico Cuenca Seminar

Nicolás Cuenca, professor Department of Physiology,Genetics and Microbiology University of Alicante Spain will be delivering a seminar, Cellular Response Associated to Retinal Diseases and Therapeutic Approaches on Monday, May 11thth at Noon in the the Moran Eye Center auditorium.


Abstract: At the cellular and molecular level, the response to retinal injury is similar in diseases like AMD, glaucoma, diabetic retinopathy and retinitis pigmentosa. All exhibit a set of cell signals that lead to controlled cell death and retinal remodeling, inflammatory responses, oxidative stress and activation of apoptotic pathways. In our hands anti-apoptotic compounds such as TUDCA and proinsulin and an3oxidant like safranal prevent retinal degeneration in animal models. In addition using Human CNS stem cells (HuCNS-SCs) we have been able to protect photoreceptors and preserve retinal integrity in a rat model of AMD. Such approaches could potentially delay retinal degeneration in human retinal diseases.